Уважаемые коллеги, доброго времени суток! Представляем вам ирландское научное издание Journal of Reproductive Immunology. Журнал имеет второй квартиль, издаётся в Elsevier Ireland Ltd., его SJR за 2022 г. равен 0,801, импакт-фактор 3,993, печатный ISSN - 1872-7603, электронный - 0165-0378, предметные области - Репродуктивная медицина, Родовспоможение и гинекология, Иммунология и аллергические реакции, Иммунология. Вот так выглядит обложка:
Здесь три редактора - Джоанне Квак-Ким, контактные данные - joanne.kwakkim@rosalindfranklin.edu,
Хироаки Шибахара - sibahara@hyo- med.ac.jp
и Джулия Сцекерес-Барто - szekeres.julia@pte.hu.
Журнал связан с Европейским обществом репродуктивной иммунологии и Международным обществом иммунологии репродукции. Цель состоит в обеспечении важнейшего форума для распространения результатов высококачественных исследований во всех аспектах экспериментальной, животной и клинической репродуктивной иммунобиологии. Это охватывает нормальные и патологические процессы:
- Мужских и женских репродуктивных путей;
- Гаметогенеза и эмбриогенеза;
- Имплантации и развития плаценты;
- Беременности и родов;
- Молочной железы и лактации.
Адрес издания - https://www.sciencedirect.com/journal/journal-of-reproductive-immunology
Пример статьи, название - Spontaneous preterm birth with placental maternal vascular malperfusion is associated with cardiovascular risk in the fifth decade of life. Заголовок (Abstract) - Women with a history of spontaneous preterm birth (SPTB) have a mildly elevated cardiovascular risk (CVR) later in life and women with a history of preeclampsia have a highly elevated CVR. In placentas of women with preeclampsia pathological signs of maternal vascular malperfusion (MVM) are often seen. These signs of MVM are also seen in a substantial part of the placentas of women with SPTB. We therefore hypothesize that in women with a history of SPTB, the subgroup with placental MVM has an elevated CVR. This study is a secondary analysis of a cohort study including women 9–16 years after a SPTB. Women with pregnancy complications known to be associated with CVR were excluded. The primary outcome was hypertension defined as blood pressure ≥ 130/80 mmHg and/or treatment with antihypertensive medication. Secondary outcomes were mean blood pressure, anthropometrics, blood measurements including cholesterol and HbA1c, and creatinine in urine. Placental histology was available in 210 (60.0%) women. MVM was found in 91 (43.3%) of the placentas, most often diagnosed by the presence of accelerated villous maturation. Hypertension was diagnosed in 44 (48.4%) women with MVM and in 42 (35.3%) women without MVM (aOR 1.76, 95% CI 0.98 – 3.16). Women with a SPTB and placental MVM showed significantly higher mean diastolic blood pressure, mean arterial pressure and HbA1c approximately 13 years after delivery, compared to women with a SPTB without placental MVM. We therefore conclude that placental malperfusion in women with a SPTB might differentiate in CVR later in life.
Abbreviations: AVM; accelerated villous maturation; CHD; coronary heart disease; CVD; cardiovascular disease; CVR; cardiovascular risk; FGR; fetal growth restriction; FRS; Framingham Risk Score; HDP; hypertensive disorders of pregnancy; MVM; maternal vascular malperfusion; PE; preeclampsia; PPROM; preterm premature rupture of membranes; SCORE; Systematic COronary Risk Evaluation; SPTB; spontaneous preterm birth
Keywords: Cardiovascular risk; Cardiovascular disease; Spontaneous preterm birth; Placental histology; Maternal vascular malperfusion