And then what is there? In fact, there are two different views on the same process in the thyroid gland with the participation of the immune system. You are told that Autoimmune thyroiditis (AIT) is an immune aggression, leads to inflammation of the gland and there is no cure? There is another scientific opinion and recovery results.
For the diagnosis of the thyroid gland, a mandatory minimum is a blood test and an ultrasound scan.
Autoimmune thyroiditis (AIT) is the most common idea of why immune cells (lymphocytes) that secrete antibodies to the thyroid gland are present in the gland. She captured the minds of doctors from different countries. Hundreds of thousands of articles have been written about her. Therefore, it seems that this is the only correct teaching. Moreover, if leading endocrinologists defend it.
There are two types of AIT. The first (most common) refers to hypothyroidism and hyperthyroidism, in which the gland tissue is depleted in exactly the same way. Antibodies to TG (thyroglobulin) and TPO (thyroperoxidase) are involved in this process of cell depletion and death.
The second variant of AIT refers only to diffuse hyperthyroidism, in which the amount of antibodies to rTSH (thyroid stimulating hormone receptor) can be increased in the blood. It is believed that AT-rTTG have a stimulating effect on thyroid cells and leads to overproduction and excess hormones (hyperthyroidism).
Autoimmune thyroiditis is contradicted by many facts that try not to notice, so as not to destroy the hypothesis of AIT. For example, in the form of a blood test, everyone can see the norm of antibodies to TPO and TG, which indicates the initial absence of immune aggression (aggressive substances can not be normal in any quantity). Improving living conditions leads to a decrease in antibodies and regenerative restoration of gland tissue (according to ultrasound).
Yes, in the thyroid gland during its overstressed activity for the formation of the right amount of hormones (under the influence of TSH and the nervous system) cells are depleted and then die. This happens with the usual activity of the thyroid gland, but in small quantities.
To remove dead cells from the gland tissue, the immune system uses antibodies to thyroglobulin (AT-TG), and antibodies to thyroid peroxidase (AT-TPO), which have a temporarily preserving (blocking, preserving) effect, apply antibodies to depleted cells. This happens in a healthy thyroid gland. Therefore, normally there is a small amount of antibodies, and with excessive congestion of the gland and its greater depletion, the number of antibodies that help preserve depleted cells and remove dead tissue elements naturally increases.
The opinion that the immune system has a useful auxiliary effect in the thyroid gland was formed by some leading experts back in the 60s, when the hypothesis of autoimmune aggression against all organs, including the thyroid gland, also emerged. But rational knowledge has been squeezed out of the scientific spotlight by increased interest in the assumption of the likelihood of autoimmune aggression.
Our Clinic offers to obtain knowledge about another scientific understanding of immune events in the thyroid gland from a special film about AIT. You will find out that in reality there is an Autoimmune process, not an Autoimmune thyroiditis.
Thyroid Clinic Dr. A.V. Ushakova wishes health to all our readers!
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