The determination of fear is one of the main examples of behavioral microbiology and a source of deep understanding of learning and memory, emotional psychology and microbiology. It is also widely considered to be a model of the pathogens of anxiety in a psychopathological model based on quality and stress.
Based on the obvious contradiction between the adaptability of conditional fear and the dysfunction of pathological anxiety, we propose a criticism of human conditional fear as an experimental model of psychopathology. We discuss trends that incorporate human behavior as an important indicator of fear and focus more on “weak” (i.e., ambiguous) rather than “strong” fear training situations (such as those included in selective training), thereby expanding the potential benefits of the human fear paradigm. We provided preliminary data to shed light on these ideas and discussed the importance of coping with systemic differences in understanding the understanding of individual susceptibility differences for pathological anxiety.
Characteristics
► It is widely believed that adjusting fear is a model of the pathogens of anxiety disorders. We discuss some limitations of the human fear paradigm in this regard.
► We recommend assessing or having a trend of action as a key component of fear.
► We believe that conditional research should rely more on indirect indicators of fear.
► We recommend focusing on the weak situation, especially the selective training paradigm.
Pavlov's definition of fear is one of the most successful laboratory examples in the history of experimental psychopathology. According to the model of the appetite regulation procedure proposed by Pavlov, it is necessary to repeatedly match the initially neutral stimulus (adjusted stimulus or CS; for example, tone) with an essentially annoying stimulus (unconditional). Stimulation or ultrasound, such as skin electrical stimulation).
As a result, the performance of CS often causes a variety of reactions that express fear. In animals, these reactions may include interruption of all motor and body movements during CS performance, suppression of ongoing tool behavior and frightening reflexes caused by loud, frightening auditory probes; and vice versa. In humans, in addition to the physiological indicators of fear (for example, increased skin conductance during CS presentation), some of which are widely used in animals, the experimenter also An oral report can be assessed by the Conference of the Parties at the time of presentation of the fear.
This basic process is an important example of behavioral and cognitive (neural) science. It can be said that many of the things we know today about fear, general learning and memory, and the study of fear are the result of somehow applying the basic paradigm that defines fear. It has proven to be not only a very useful tool for revealing the psychological processes that control the generation and expression of fear, but also for emotions and shared memory functions, but also for studying the neurological basis of emotions and learning.
The idea is that regulating fear provides a laboratory model for the pathogens of anxiety disorders in the real world.
According to this view, pathological anxiety of essentially harmless stimuli (such as home spiders or crowded places) may develop with unpleasant events or traumatic experiences (such as frightened mothers or panic attacks); such connections can be personal Experience, you can also choose. Like COP, these initially harmless stimuli can cause excessive fear or anxiety and encourage behavior by referring to terrible events associated with them.
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